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With great interest we read the extensive review on JAK kinase targeting in hematologic malignancies by Springuel, Renauld and Knoops.[1][1] It provides a clear overview of the plethora of cytokine receptor complexes and downstream signaling adapter molecules that are mutated in hematologic

Structural modeling of JAK1 mutations in T-cell acute lymphoblastic leukemia reveals a second contact site between pseudokinase and kinase domains