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The neoplastic B-cell derived Hodgkin and Reed-Sternberg cells (HRS) in classical Hodgkin's lymphoma (cHL) undergo a substantial loss of B-cell identity. It has been suggested that epigenetic mechanisms such as DNA methylation are involved in this cHL-associated reprogramming. [1][1] In a recent

ETS1 encoding a transcription factor involved in B-cell differentiation is recurrently deleted and down-regulated in classical Hodgkin's lymphoma