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We recently published in this journal an overview of the currently known genetic events required for the development of pediatric acute myeloid leukemia (AML).[1][1] These aberrations can be subdivided into type-I aberrations that result in uncontrolled proliferation, and type-II aberrations that

Low frequency of type-I and type-II aberrations in myeloid leukemia of Down syndrome, underscoring the unique entity of this disease