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Systemic amyloidosis (AL) is a plasma cell dyscrasia in which the clone secretes free kappa (κ) or lambda(λ)-immunoglobulin light chains (FLCs).[1][1] These light chains do not fold into the proper tertiary conformation and form protein deposits, causing organ damage.[2][2] The most commonly

haematologica

Amyloidosis relapsing after autologous stem cell transplantation treated with bortezomib: normalization of detectable serum-free light chains and reversal of tissue damage with improved suitability for transplant