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Treatment of plant tissue with hyphal wall components (HWC) is known to induce accumulation of phytoalexins1,2,3,4). In previous studies5,6,7,8,9,13), the present authors reported that rishitin was synthesized in healthy tissue neighbouring the infected lesion, transported into necrotic tissue and accumulated there. Rishitin, remaining in healthy tissue surrounding the necrotic lesion, was supposed to be transformed rapidly to rishitin-M-1, rishitin-M-2 and more hydrophilic unknown compounds in succession, resulting in accumulation of trace amounts of rishitin in the healthy tissue zone8,9,15). Accordingly, a question raised as to whether HWC of Phytophthora infestans caused necrotic lesion in the tuber tissue, resulting in accumulation of rishitin, or induced this accumulation without causing necrosis. Tubers of the potato cultivar Rishiri with R1-gene stored in a dark room at 4C were used. The tubers were sterilized with 1% sodium hypochlorite, washed with water and then sliced into disks 16mm in diameter and 1mm thick. The disks were

Relation between necrosis and rishitin accumulation in potato tuber slices treated with hyphal wall components of phytophthora infestans.