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Essential fatty acids (EFAs) are essential components of structural phospholipids in all tissues and modulate cell membrane fluidity and functions. The availability of long chain poly unsaturated fatty acids (PUFA) (>18 carbon atoms), such as arachidonic and docosahexaenoic acids, is important for early human growth and development of membrane rich tissues such as brain and retina [1]. Moreover, w-b and co-3 PUFA serve as precursors of eicosanoids with important biological roles as mediators of immune and vascular functions as well as platelet aggregation [2, 3]. It was hypothesized that children with cholestatic liver disease have a poor PUFA status, because bile acids contribute to efficient PUFA absorption from the gut and because long chain polyunsaturated (LCP) fatty acids are synthesized from their precursors, mainly in the liver by desaturase and elongase systems that are adversely affected *Correspondence to: H .H. Fouad

Essential Fatty Acids Status in Infants and Children with Chronic Liver Disease