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Background The portal venous system is a major vein that leads to the liver. Portal hypertension may be developed by intrinsic liver disease, obstruction, or structural changes that result in increased portal venous flow or increased hepatic resistance. Usually, vascular channels are smooth, but liver cirrhosis can cause to become irregular and tortuous with accompanying increased resistance to flow. This increased pressure resulting in varices or dilations of the veins and tributaries. Pressure within the portal system is dependent upon both input and blood flow in the portal vein, and hepatic resistance to outflow. Normally portal vein pressure ranges between 1-4 mm Hg higher than the hepatic vein free pressure, and not more than 6 mm Hg higher than right atrial pressure. Pressure that exceed these limits define portal hypertension.

Portal hypertension, cirrhosis of liver and the unsuspected intrinsic property of melanin to dissociate the water molecule, like chlorophyll in plants