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and Implications The objective of this study was to investigate the nature of the effect of the F94L variant in the myostatin (MSTN) gene on economic traits in Limousin and Limousin-Angus crossbred animals in the context of genomic analyses using Illumina BovineSNP50 Bead chip genotypes. Introduction Mutations in the MSTN gene have been identified at high frequencies in some heavily muscled beef cattle breeds. The mutations comprise deletions, insertions, or nucleotide substitutions that interrupt production or activity of the MSTN gene. There are at least nine identified mutations that cause muscular hypertrophy. The F94L mutation in MSTN is common in purebred Limousin and descendant crosses such as Lim-Flex. Lim-Flex is a cross between Limousin and (Red or Black) Angus, and is a popular Limousin hybrid in the United States. In this research, we characterize the effect of the F94L mutation on commonly recorded traits in Limousin cattle.

Characterization of the F94L Double Muscling Mutation in Pure- and Crossbred Limousin Animals