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Serum and tissue homogenates of lung and kidney from 264 mice, half of which had been exposed to continuous or intermittent nitrogen dioxide (NO 3  at levels of 0.5 ppm, 0.6 ppm, and 0.8 ppm in three independent experiments, were assayed for intravenously introduced horseradish peroxidase (HRP), a molecular probe for protein leakage. Disc gel elec-trophoresis and enzyme kinetic assays were used independently to quantitate HRP content after 3 and 6 weeks of NO 2  exposure, and at 5.5 hr after HRP injection. Of 6 test periods, 5 showed a greater lung HRP content for the NO 2 , exposed animals by gel scan analysis, and 3 of the 5 increases were statistically significant (p &gt;.05, p &gt;.025, and p &gt;.0025). Similar trends were noted with the HRP kinetic assay. Serum and kidney comparisons showed no consistent differences; 1 of 6 test periods for each was statistically significant. The findings implicate an NO 2 , induced overload of the bidirectional protein transport system of the lung.

Serum and Lung Clearance of Exogenous Horseradish Peroxidase: Influence of Low Levels of Nitrogen Dioxide