English (United Kingdom)

https://curated-unify.zendy.io/wp-json/zendy-region/v1/featured_content/oa?rat=en

https://curated-unify.zendy.io/wp-json/zendy-region/v1/highlighted_journal/

Global: Zendy Plus annual

Presents the access of premium content as premium feature

Premium Content

Presents the keyphrase highlighting as premium feature

Keyphrase Highlighting

Presents the summarisation as premium feature

Summarisation

Insights

Presents the pdf analysis as premium feature

PDF Analysis

Presents the zaia usage as premium feature

ZAIA

Global: Zendy Tools annual

Global: Zendy Free Plan

Global: Zendy Tools monthly

Global: Zendy Plus monthly

Midkine (MK), a heparin-binding cytokine, is considered to be involved in disease mechanisms of several autoimmune (e.g rheumatoid arthritis, systemic lupus erythematosus and multiple sclerosis) and autoinflammatory (e.g Crohn’s disease and ulcerative colitis) diseases. Behçet’s syndrome (BS) is accepted as a mixed pattern disease with evidence of both acquired autoimmune component and autoinflammatory components. Therefore, our hypothesis is that MK might be overexpressed in BS patients and if so, it might serve as a disease marker in BS. Furthermore, inhibition of the hypothesized MK overexpression using MK inhibitors such as MK-aptamer might contribute to the management of BS.

A Possible Role for Midkine in the Pathogenesis of Behçet’s Syndrome