English (United Kingdom)

https://curated-unify.zendy.io/wp-json/zendy-region/v1/featured_content/oa?rat=en

https://curated-unify.zendy.io/wp-json/zendy-region/v1/highlighted_journal/

Global: Zendy Plus annual

Presents the access of premium content as premium feature

Premium Content

Presents the keyphrase highlighting as premium feature

Keyphrase Highlighting

Presents the summarisation as premium feature

Summarisation

Insights

Presents the pdf analysis as premium feature

PDF Analysis

Presents the zaia usage as premium feature

ZAIA

Global: Zendy Tools annual

Global: Zendy Free Plan

Global: Zendy Tools monthly

Global: Zendy Plus monthly

Hypereosinophilia (HE) is defined as a persistent increase in absolute eosinophil counts (AEC) to levels >1.5 × 109/L. HE can be ‘primary’ or ‘secondary’, more often accompanying infections, allergic disorders, autoimmune diseases or myeloproliferative/lymphoproliferative neoplasms [1,2]. In 2016, the WHO revised the classification of eosinophilic disorders, distinguishing myeloid/lymphoid neoplasms with eosinophilia and recurrent genetic lesions (such as rearrangement of PDGFRA, PDGFRB, FGFR1 and PCM1-JAK2), chronic eosinophilic leukemia (CEL) not otherwise specified (NOS), and idiopathic HE [3,4].

Sorafenib Induced Complete Cytogenetic and Molecular Response in a Chronic Eosinophilic Leukemia Case with t(12;13) Translocation