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Low-dose radiation in apolipoprotein E-deficient (ApoE -/- ) mice has a protective effect with less subsequent atherosclerosis. Inflammation and apoptosis play major roles in the development of atherosclerosis. We evaluated the temporal pattern of the development of histologic atherosclerosis, inflammation with 18 F-FDG, and apoptosis with 99m Tc-rhAnnexin V-128 at 3 time points.

the journal of nuclear medicine

Evaluation of Apoptosis with <sup>99m</sup>Tc-rhAnnexin V-128 and Inflammation with <sup>18</sup>F-FDG in a Low-Dose Irradiation Model of Atherosclerosis in Apolipoprotein E–Deficient Mice

Evaluation of Apoptosis with 99mTc-rhAnnexin V-128 and Inflammation with 18F-FDG in a Low-Dose Irradiation Model of Atherosclerosis in Apolipoprotein E–Deficient Mice

Evaluation of Apoptosis with ^99mTc-rhAnnexin V-128 and Inflammation with ¹⁸F-FDG in a Low-Dose Irradiation Model of Atherosclerosis in Apolipoprotein E–Deficient Mice