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COVID-19 as many other virus infections produces oxidative stress, i.e. superiority of free radicals over antioxidants. Free radicals have one or more non-pared electrons, which attracts another electron from the surrounding. So the substrate is oxidised (lipoperoxidation, oxidation of proteins, of DNA and RNA, etc.). Sometimes we use the expression ROS (reactive oxygen species) which except free radicals includes substances which don ́t correspond we the de inition of free radicals, but act like them (singlet oxygen, ozone, hydrogen peroxide). The reason of these virus reactions are energy pro it and penetration through the membranes into tissues (sinuses, lungs, kidneys etc., harmfully acts on lymphatic and vascular endothel). Free radicals in WBC ́s kill bacterias, but on virus infections have opposite in luence. Oxidation of biomoleculs depends on redox potential. Substance with higher redox potential can oxidise substances with lower potential which have prooxidative action. Redox potential of antioxidants must be suf iciently low, otherwise they will act proxidatively. Virus is under different pressures. Evolutionarily convenient is for him not to kill to much patients and so infected people can disseminate viruses and on the other hand is the pressure to maintain mutations, which will improve the ability of transmission. The amount of COVID-198 virusis during the irst week is high, the second week it decreases, therefore the therapy must start quicly.

Coronaryvirus infection, oxidative stress in ENT