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weeks increased the number of arriving to drinking-bowl in 1,4 times (p<0,02), and possibly increased the number of drunk sucrose and percentage of consumed sugar to total number of liquid with experimental pathology. Preventive and therapeutic use of derivative 2-oxoindoline 18 effectively countered anhedonia, and increase of the number of arriving to drinking-bowl with sucrose 1,4 times (p<0,02) and increase of drunk sucrose 1,3 times (p<0,02) in comparison with control pathology without correction. Also, substance 18 assisted in the advantage of sucrose solution. Also, during experimental neurosis in the test sucrose consumption advantage, the percentage of drunk water with sugar in comparison with control group was decreased. After 4 weeks of neurosis, diazepam intake increased the number of arriving to drinking-bowl in 1,4 times (p<0,02), and possibly increased the number of drunk sucrose and percentage of consumed sugar to total number of liquid in comparison with such during experimental pathology. Preventive and therapeutic use of amide of 2-oxoindoline-3-glyoxylic acid with substance 18 after 4 weeks of neurosis modeling effectively withstood anhedonia. During this period the substance possibly assists in the advantage of sucrose among water. So, experimental neurosis is accompanied with the development of typical emotional and behavioral disorders (decrease of quantative and qualitative indices of sucrose consumption). Preventive and therapeutic use of amide of 2-oxoindoline-3-glyoxylic acid effectively corrected the development of taste disorders in sucrose consumption and it didn’t exceed diazepam. Experimental neurosis caused the development of anhedonia. Therapeutic and preventive use of the substance 18 effectively decreased the manifestations of anhedonia after experimental neurosis in rats. Such results can be essential for further investigation of monoaminergic mechanisms of the derivatives of 2-oxoindoline-3-glyoxylic acid during neurotic pathology in animals.

RELIABILITY OF URINE AS SOURCE FOR BIOLOGICAL INFORMATION FOR RISK ESTIMATION FOR PROSTATE MALIGNANCY