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The Adenomatoid odontogenic tumour is considered as a benign, nonneoplastic,non invasive, lesion with a slow but progressive growth [1]. Philipsen and Birn suggested the most widely accepted abbreviated terminology for Adenomatoid odontotgenic tumour, as AOT [2]. Various names have been used in the literature for AOT like adenoameloblastoma, ameloblastic adenomatoid tumour, adamantinoma, epithelioma adamantinoma, or teratomatous odontoma [3]. AOT is an uncommon tumour of odontogenic origin, accounting for 2.2-7.1 % of all odontogenic tumours [4]. The lesion was initially described by Steensland in 1905 and later further described by Philipsen and Reichart in 1907 as they described three clinical-topographic variants of AOT: follicular (71%), extrafollicular (23%) and peripheral (4%). The follicular and extra follicular occur in intrabony sites while peripheral variant occurs in the gingival issue of the tooth bearing area [4].

Hybrid Variant of Adenomatoid Odentogenic Tumour