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Thrombocyte and Erythrocyte Indices in Sepsis and Disseminated Intravascular Coagulation Sepsis is the inflammatory response against infection. The existence of DIC during sepsis indicates a poor prognosis and coagulation abnormalities and thrombocytopenia may exist. The aim of this study was to investigate platelet and erythrocyte indices in sepsis patients with DIC and without DIC. In both groups coagulation tests, platelet count and indices, erythrocyte count and indices were retrospectively analysed. In the sepsis plus DIC patients the prothrombin time and D-dimer values were found significantly higher and fibrinogen, platelet and plateletcrit were found significantly lower than in the sepsis without DIC group. The analysis of mean platelet volume, platelet distribution width, erythrocyte count and indices revealed no significant differences between the two groups. These results showed us that the depression of bone marrow in septic patients with DIC and without DIC did not differ. The activation of the coagulation system might probably be the cause of thrombocyte depletion in DIC. Trombocitni i Eritrocitni Indeksi u Sepsi i Diseminovanoj intravaskularnoj Koagulaciji Sepsa predstavlja inflamatorni odgovor na infekciju. Prisustvo diseminovane intravaskularne koagulacije (DIK) tokom sepse ukazuje na lošu prognozu, a mogu se javiti i poremećaji u koagulaciji i trombocitopenija. Cilj ove studije bio je da se ispitaju trombocitni i eritrocitni indeksi kod pacijenata sa sepsom i DIK, odnosno sepsom bez DIK. U obe grupe retrospektivno su analizirani testovi koagulacije, broj i indeks trombocita i broj i indeks eritrocita. Kod pacijenata sa sepsom i DIK, protrombinsko vreme i vrednosti D-dimera bili su značajno povišeni, dok su fibrinogen, trombociti i trombocitokrit bili značajno niži nego u grupi sa sepsom bez DIK. Prilikom analize srednje zapremine trombocita, širine distribucije trombocita i broja i indeksa eritrocita nisu otkrivene značajne razlike između dve grupe. Ovakvi rezultati ukazuju na to da kod pacijenata sa sepsom i DIK i pacijenata sa sepsom bez DIK nema razlika u depresiji koštane srži. Aktivacija koagulacijskog sistema predstavlja mogući uzrok manjka trombocita u DIK.