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It is believed that an organism remains normoglycemic despite an increase in the beta-cell mass because of decreased insulin production by beta-cells on a per-cell basis. However, some transgenic mouse models with beta-cell hyperplasia suggest that insulin production remains excessive and that normoglycemia is maintained by insulin resistance.

diabetes

Increased β-Cell Mass by Islet Transplantation and <i>PLAG1</i> Overexpression Causes Hyperinsulinemic Normoglycemia and Hepatic Insulin Resistance in Mice

Increased β-Cell Mass by Islet Transplantation and PLAG1 Overexpression Causes Hyperinsulinemic Normoglycemia and Hepatic Insulin Resistance in Mice