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low concentra-tions of zinc enhanced, whereas higher concentrations de-creased, the magnitudes of the voltage-activated hemichan-nel currents. The present study was designed to determinethe effects of zinc on hemichannels formed by Cx26, aconnexin reportedly expressed on dendrites of carp hori-zontal cells and implicated in a mechanism for photorecep-tor feedback (2, 3, 4). In addition, we examined whetherhistidine, a zinc chelator, would block the action of zinc onCx26 hemichannel currents, or would exert a direct effecton those currents.

Histidine Suppresses Zinc Modulation of Connexin Hemichannels