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In a previous paper, several theories as to the etiology of hemiplegic amyo trophy and its mechanisms were introduced and discussed.' From the author's investigations, another possible explanation for the occurrence of hemiplegic amyotrophy is, in addition to the vasomotor theory, that loss of control from the central trophic system of the muscle and/or peripheral nerves (where a cen tral lesion might be) induces muscle wasting and/or denervation of the lower motor neurons, thus secondarily contributing to muscle atrophy. Recently, some investigators have reported peripheral nerve lesions in the involved extremities of hemiplegic patients, resulting from the deprivation of central nervous system control2,3,7-s However, other examiners hold these results in question?-6,10,11 By measuring the maximum motor nerve conduction velocity (MCV) of the ulnar nerves in the involved and uninvolved extremities of hemiplegic patients, the author attempted to determine whether lower motor neurons were involved in the development of hemiplegic amyotrophy. On the whole, results of the MCV study of 86 cases revealed a statistically significant lower motor nerve conduction velocity of the ulnar nerve in involved extremities as compared to uninvolved extremities. In further investigating the results, the author correlated the motor conduction velocities of ulnar nerves with clinical data.

HEMIPLEGIC AMYOTROPHY AND MOTOR NERVE CONDUCTION VELOCITY IN HEMIPLEGIC PATIENTS