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The most relevant mycotoxin from the trychotecene group, T-2, causes  prominent citotoxic effects. The toxin is a secondary product of fungi from  the genus Fusarium that contaminates feed. Oraly intaken, T-2 is absorbed  fast in the upper digestive system and within only 3 to 4 hours later reaches  liver, kidneys and muscle tissue. Clinical and pathological changes are  sometimes not obvious. The case of mycotoxicosis in a breeder flock of  chickens, here presented, is aimed to underline the significance of clinical  and pathological diagnosis supported with laboratory analysis that gave an  objective causative diagnosis. On the farm, the disease occurred suddenly and  with total cessation of feed consumption. First cases were recorded in the  flock at the age of 42 weeks. Grouping, intensive breathing and lying with  overstretched legs and extended neck were symptoms observed in birds. Evident  necrosis of beak tips and painful multi-focal necrosis in oral cavity were  recorded during the clinical examination. On section, dark unclothed blood  was first observed. Other postmortem findings included: filled gizzard with  mucosal erosions and easy-removable cuticle, enlarged congested liver with  multi-focal necrosis and subcapsulary bleeding. The mortality rate increased  by 4%, and the drop of laying rate was by about 18%. The fertility rate  decreased by 22%. There was the increased number of rejected hatching eggs,  12%. Culture of the complete diet resulted in approximately 150000 colonies  per 1g of Fusarium. T-2 was detected by using ELISA in concentration of 480  μg/kg, which corresponded to the upper limit of maximum permitted  concentrations for chickens, according to national legislations. This bylaw  interpretation of “tolerable” concentrations of mycotoxins provokes  controversy among experts and public. [Projekat Ministarstva nauke Republike  Srbije, br. TR 31071

Clinical and pathomorphological diagnostics of mycotoxicosis in parent poultry flock caused by T-2 trychotecene