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Various factors have been suggested in the pathogenesis of feline resorptive  lesions, such as periodontal disease, dietary factors, mechanical stress,  developmental tooth defects, breed and viral disease, although none of these  factors have been definitively proven to be the direct cause. It was recently  published that normally enamel in cats is significantly thinner at the  cemento-enamel junction, and both enamel and dentine are significantly less  mineralized than elsewhere on the tooth. However, it is still unclear what  anatomical features of the tooth surface are associated with a predisposition  for resorptive lesions, and what is the initiating cause for the clastic  activity afterwards. The present study was undertaken with the aim to  describe enamel properties of transport and distribution of organic molecules  in intact feline teeth and teeth affected with resorptive lesions. The  results indicate that damaged enamel is prone to a greater bilateral  diffusion process, leading to continuous disruption of the enamel structure.  Also, teeth that are subjected to occlusal stress are at greater risk of  destruction because micro fractures produce disarrangements in feline dental  tissue diffusion homeostasis. The relationship between these features with  feline dental resorptive lesions requires further studies

Identifying enamel diffusion properties in feline teeth affected with resorptive lesions