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Abuse of anabolic-androgenic steroids (AAS) produces side effects in   different tissues, with oxidative stress linked to their pathophysiology,   being involved in fibrosis, cellular proliferation, and tumorigenesis. The   aim of this study was to examine the acute effects of nandrolone decanoate   (ND) on oxidative stress in isolated rat heart. The hearts of male Wistar   albino were excised and perfused according to the Langendorff technique at   gradually increasing coronary perfusion pressures (40-120 cmH2O). The hearts   were perfused with ND at doses of 1, 10 and 100 μM. Oxidative stress markers,   including the index of lipid peroxidation (thiobarbituric acid reactive   substances (TBARS)), nitric oxide (nitrites; NO2-), the superoxide anion   radical (O2-) and hydrogen peroxide (H2O2) were measured in the coronary   venous effluent. Our results showed that acute effects of ND do not promote   the production of reactive oxygen species (ROS). Our finding pointed out that   the highest concentration of ND may even possess some anti-oxidative   potential, which should be examined further

Acute effects of nandrolone decanoate on oxidative stress in isolated rat heart