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Tuberculosis (TB) is a devastating disease that leads to 10 million deaths per year worldwide.1 Despite enormous advances in TB research in recent years, the surge of drug-resistant strains and deadly synergy with the HIV virus have threatened to destabilise gains made in its control. During my career as a microbial geneticist, I have studied the physiology of Mycobacterium tuberculosis, the causative agent of TB, and its interaction with the human macrophage, its primary host cell. The major function of the macrophage is to engulf and destroy invading organisms, but many pathogens, such as TB, have developed intricate and clever ways to avoid or subvert these host defences.

The Super TB Experiment: Evolution and Resolution of an Experiment with Dual-Use Concerns