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In the model plant Arabidopsis thaliana, meristem acquisition of inflorescence fate promotes both the production of flowers at the expense of leaves and the elongation of internodes to generate the inflorescence. Competence to respond to floral-inductive signals requires the activities of two BELL homeodomain proteins: PENNY WISE (PNY) and POUND-FOOLISH (PNF). Inactivation of these genes blocks meristem conversion to an inflorescence meristem fate, but their mode of action remains unclear. My data provide evidence that the ectopic activity of four lateral organ boundaries genes acting in the same pathway blocks the conversion to IM fate in pny pnf double mutants. This pathway is defined by BLADE-ON-PETIOLEl/2 (BOPl/2), ARABIDOPSIS THALIANA HOMEOBOX GENE1 (ATH1), and KNOTTED1-LIKE FROM ARABIDOPSIS THALIANA6 (KNAT6). My data are consistent with the model that ectopic BOPl/2 activity in meristems promotes the misexpression of ATH1 and KNAT6 whose products form a transcription factor complex that antagonizes competence to flower.

Genetic analysis of co-ordination of flowering and regulation of Inflorescence Architecture in Arabidopsis thaliana