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The intestinal motility induced by occlusion of the cranial mesenteric artery was studied on unanesthetized spinal dogs.1. An increase in intestinal motility and tone occurred most frequently (in 23 out of 32 observations) in the initial phase of arterial occlusion, while a decrease occurred less frequently (in 2 out of 32 observations).2. This increase in intestinal motility and tone was abolished by tetrodotoxin and atropine, suppressed by pentobarbital, potentiated by physostigmine, and remained unchanged with vagotomy or hexamethonium.3. From these results it is suggested that the release of acetylcholine through excitation of the intramural cholinergic neurons due to oxygen lack or an increase in CO2 during arterial occlusion is responsible for the increased intestinal motility.4. It is discussed concerning a possible mechanism operating in the decreased intestinal motility by arterial occlusion.

CHOLINERGIC MECHANISM FOR INTESTINAL HYPERMOTILITY CAUSED BY ARTERIAL OCCLUSION