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disease. In recent three years, she had been treated with 20mg of hydrocortisone per day. Plasma cortisol levels before hydrocortisone administration were very low and urinary 17KGSlevels did not respond to ACTH-Z. Plasma ACTHlevels before hydrocortisone administration were high. Peripheral thyroid hormone levels were normal but response of plasma TSHto TRHinjection was high and prolonged. 131I-uptake in thyroid gland was very high. Thyroid gland aspiration biopsy revealed infiltrations of many lymphocytes in the gland. Plasma gonadotrophins levels were very high and response to synthetic LH-RH were excessive. Thyroglobulin antibody and thyroid microsomal antibody were found in her sera. Specific adrenal antibody against zona fasciculata cells of dog adrenal cortex was also found by immunofluorescence method. Ovarian antibody, parietal cell antibody and antinuclear factor were not detected. Leukocyte mygration inhibition test using rabbit adrenal tissue was normal. These findings indicate that her goiter was caused by chronic thyroiditis and oligomenorrhea was caused by ovarian failure.

A CASE OF IDIOPATHIC ADDISON'S DISEASE WITH CHRONIC THYROIDITIS AND OVARIAN DYSFUNCTION