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To determine whether protein kinase C-iota (PKC-iota) is associated with glucose metabolism in non-small-cell lung cancer (NSCLC) and whether its regulatory effect on metabolic and biological changes observed in NSCLC can be mediated by glucose transporter 1 (GLUT1).

<p>Protein kinase C-iota-mediated glycolysis promotes non-small-cell lung cancer progression</p>