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Craniosynostosis is a developmental disorder characterized by the premature fusion of skull sutures, necessitating repetitive, high-risk neurosurgical interventions throughout infancy. This study used protein-releasing Titania nanotubular implant (TNT/Ti) loaded with glypican 3 (GPC3) in the cranial critical-sized defects (CSDs) in Crouzon murine model ( Fgfr2 c342y/+  knock-in mutation) to address a key challenge of delaying post-operative bone regeneration in craniosynostosis.

<p>Titania nanotube-based protein delivery system to inhibit cranial bone regeneration in Crouzon model of craniosynostosis</p>