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Nonalcoholic fatty liver disease (NAFLD) has been considered the most commonly occurring chronic hepatopathy in the world. Ghrelin o -acyltransferase (GOAT) is an acylation enzyme which has an acylated position 3 serine on ghrelin. Recent investigation revealed that activated autophagy could attenuate liver steatosis. The aim of this study was to explore therapeutic roles that inhibit GOAT exerted in NAFLD, and its potential association with autophagy.

Inhibition of ghrelin<em> o</em>-acyltransferase attenuated lipotoxicity by inducing autophagy via AMPK&ndash;mTOR pathway

Inhibition of ghrelin<em> o</em>-acyltransferase attenuated lipotoxicity by inducing autophagy via AMPK–mTOR pathway