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COPD is a leading cause of mortality worldwide, and cigarette smoke is a pivotal risk factor. Adenovirus is a common cause of acute exacerbations of COPD and expedites COPD progression. Lung stem/progenitor cells play an important role in the development of COPD, while the relevant mechanism remains elusive. Here, we investigated the number of lung CD31 - CD45 - Sca-1 + cells and sonic hedgehog (Shh) signaling pathway expression levels in cigarette smoke extract (CSE)-induced emphysema mice, as well as the relevant effects of acute adenovirus infection (AAI).

international journal of chronic obstructive pulmonary disease/international journal of copd

Changes in the number of CD31&lt;sup&gt;&amp;minus;&lt;/sup&gt;CD45&lt;sup&gt;&amp;minus;&lt;/sup&gt;Sca-1&lt;sup&gt;+&lt;/sup&gt; cells and Shh signaling pathway involvement in the lungs of mice with emphysema and relevant effects of acute adenovirus infection