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Prostate cancer (PCa) is the most common solid tumor in men and the second leading cause of male mortality in North America (1). It is increasingly obvious that chemokines within the tumor microenvironment are involved in the acquired capability for invasive growth and metastasis (2-4). Multiple chemokines such as C-C motif chemokine 2, interleukin-12, interleukin-8 (IL-8) are principle factors implicated in the progression of PCa (3,5,6). IL-8 derived either from tumor cells themselves or other cellular components is crucial for PCa progression (6-8). IL-8, also known as C-X-C motif chemokine ligand 8 (CXCL8), exerts many biological effects through its C-X-C motif chemokine receptors 1/2 (CXCR1 and CXCR2). IL-8 and its receptors were overexpressed in PCa biopsy specimens, however, they decreased in prostatic intraepithelial neoplastic (PIN) foci and even Original Article

MicroRNA-106a suppresses prostate cancer proliferation, migration and invasion by targeting tumor-derived IL-8