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Androgen depletion therapy (ADT) is performed for the treatment of prostate cancer (PCa). However, most patients with PCa will develop castration-resistant prostate cancer (CRPC) regardless of the treatment with ADT. Reactivation of androgen receptor (AR) and tumor recurrence occur in the presence of low androgen levels in patients with CRPC. Therefore, it is important to suppress the AR signal in CRPC. Based on these considerations, secondgeneration AR-targeted drugs, including abiraterone, a CYP17A1 inhibitor, and enzalutamide, a potent AR Original Article

translational cancer research

Analysis of novel enzalutamide-resistant cells: upregulation of testis-specific Y-encoded protein gene promotes the expression of androgen receptor splicing variant 7