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The end-stage liver disease causes a metabolic dysfunction whose most prominent clinical feature is the loss of skeletal muscle mass (SMM). In living-donor liver transplantation (LDLT), liver graft regeneration (GR) represents a crucial process to normalize the portal hypertension and to meet the metabolic demand of the recipient. Limited data are available on the correlation between pre-LDLT low SMM and GR.

hepatobiliary surgery and nutrition

Is there any correlation between liver graft regeneration and recipient’s pretransplant skeletal muscle mass?—a study in extended left lobe graft living-donor liver transplantation