Open AccessDistinct metabolic preference of atypical KRAS mutant
Author(s) -
Heesun Cheong
Publication year - 2020
Publication title -
annals of translational medicine
Language(s) - English
Resource type - Journals
eISSN - 2305-5847
pISSN - 2305-5839
DOI - 10.21037/atm-20-3252
Subject(s) - kras , mutant , computational biology , preference , genetics , biology , mutation , bioinformatics , medicine , gene , microeconomics , economics
Division of Cancer Biology, National Cancer Center, Goyang, Republic of Korea Correspondence to: Heesun Cheong. Division of Cancer Biology, National Cancer Center, 323 Ilsan-ro, Ilsandong-gu, Goyang, Gyeonggi-do 10408, Republic of Korea. Email: heesunch@ncc.re.kr. Provenance and Peer Review: This article was commissioned and reviewed by the Section Editor Le Li, MD, PhD (Department of Pancreatic and Biliary Surgery, The First Affiliated Hospital of Harbin Medical University, Harbin Medical University, Harbin, China). Comment on: Hobbs GA, Baker NM, Miermont AM, et al. Atypical KRAS G12R Mutant Is Impaired in PI3K Signaling and Macropinocytosis in Pancreatic Cancer. Cancer Discov 2020;10:104-23.
Accelerating Research
Robert Robinson Avenue,
Oxford Science Park, Oxford
OX4 4GP, United Kingdom
Address
John Eccles HouseRobert Robinson Avenue,
Oxford Science Park, Oxford
OX4 4GP, United Kingdom