Open AccessDioscin attenuates lipopolysaccharide-induced inflammatory myocardial injury through oxidative stress-related pathway
Author(s) -
Zheng Xu,
Xiaomin Li,
Xuewen Li,
Yuping Gao,
Xiaolong Mi
Publication year - 2021
Publication title -
annals of palliative medicine
Language(s) - Uncategorized
Resource type - Journals
eISSN - 2224-5839
pISSN - 2224-5820
DOI - 10.21037/apm-21-1613
Subject(s) - malondialdehyde , oxidative stress , lipopolysaccharide , medicine , apoptosis , superoxide dismutase , tumor necrosis factor alpha , reactive oxygen species , tunel assay , western blot , pharmacology , interleukin , glutathione , inflammation , mtt assay , immunology , cytokine , biochemistry , chemistry , enzyme , gene
Lipopolysaccharide (LPS) is one of the main causes of myocardial injury. Dioscin has a protective effect on myocardial injury induced by LPS; however, the biological function and mechanism remain unclear. The purpose of this study was to investigate the effect of dioscin on myocardial injury induced by LPS.
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