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Type 2 diabetes is a complex metabolic disorder with both genetic and environmental factors such as food habits and lifestyle contributing to its pathogenesis [1] . Due to its complex aetiology, the progress of discovery of genetic components for Type 2 diabetes had been very slow until the advent of high throughput genome-wide association (GWA) studies [2][3] . Most individuals with T2DM suffer serious complications of chronic hyperglycemia, involved in nephropathy, neuropathy, retinopathy, and accelerated development of cardiovascular disease. When the micro vascular complications are related to the degree of fasting hyperglycemia [4] , macro vascular complication like atherosclerosis is related to postprandial glycemic level [5] . One of the regulators of postprandial glucose level is GLP-1.

TCF 7l2 Gene Variant Rs7903146 Affects the Risk of Type 2 Diabetes by Modulating Incretin Secretion