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Development and progression of heart failure (HF) strongly relates to microvasculature damage, endothelium dysfunction and target organ perfusion abnormalities [1]. The conceptual molecular mechanisms of shaping vascular wall cell injury base on an opinion regarding an imbalance between a damage of target cells and prevention or restore of altered structure and impaired function of them [2]. The central player in mediating vascular tissue reparation is endogenous regenerative capacity of residential cells with pluripotent activity and predominantly originated from bone-marrow stem cells and rarely from peripheral stem cells [3].

The Endothelial Progenitor Cell Dysfunction: The Missed Predictor of Heart Failure Outcomes?