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Alzheimer's disease (AD), also known as senile dementia, is a neurodegenerative disease that occurs frequently with age. Its clinical manifestations are mainly progressive memory impairment, cognitive dysfunction, personality changes and language disorders, which frequently occur in the elderly [1] . The most important pathological feature of AD is the formation of extracellular amyloid polypeptide deposition known as senile plaques (SP) and intracellular neurofibrillary tangles (NFT) [2] . There is no effective strategy for treatment of AD at present in clinic, then, the early prevention and treatment of Abstract:

Effect of SBF on Aβ25-35 Induced- Abnormal Changes of PKA and tau Protein Phosphorylation in N2a Cells