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Currently antitumor therapies consist in the neoplasic cells attack by chemotherapy and radiotherapy. Successful treatment depends on the type of cancer, because each cancer has particular characteristics of survival and immune evasion. The neoplasic cell biology studies demonstrated a persistent immunosuppressive environment surrounding transformed cells. Thus, compounds that are capable of acting as agonists of Toll-like receptors (TLRs) may represent promising candidates to be developed as medicaments against cancer. Thus, the general objectives of this study are to characterize and compare the histologic effects of P-MAPA associated with the chemotherapeutic agent Gemcitabine to treat pancreatic cancer in rats.

EFFECTS OF P-MAPA ASSOCIATED WITH GEMCITABINE IN THE PANCREATIC CANCER: HISTOLOGIC EFFECTS