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For years, scientists and clinicians believed that systole was the cornerstone mechanism of heart functioning. As a result, all treatments for heart failure (HF) were directed at treating systolic dysfunction. But the heart is actually in diastole two-thirds of the time. In the 1980s, the medical community began to recognize that some patients with a normal ejection fraction were experiencing signs and symptoms of heart failure. Scientists evaluated these patients and found that even though systolic function was adequate, diastolic function was suboptimal. At this time, diastolic dysfunction was deemed to be clinically significant [1].

The Correlation between the Plasma Level of NT-Pro BNP and Left Ventricular Diastolic Dysfunction