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Acute kidney injury (AKI), being a common complication of various conditions, is associated with high mortality and became a substantial challenge for modern medicine [23]. AKI is known to be a risk factor for development and progression of chronic kidney disease (CKD) that represents serious medical and social problem [7]. Conventional criteria for AKI diagnostics and staging, according to current clinical practice guidelines, are serum creatinine concentration (PCr) elevation and decline of urine output [2]. These criteria are also used for AKI diagnostics in patients undergoing hematopoietic stem cell transplantation (HSCT) [9,18]. However, fluctuations of PCr and urine output, reflecting development of AKI, are not reliable markers of earlier, potentially reversible stages of tubular damage in patients following HSCT [4, 8, 12]. Vladimir A. Dobronravov1, 2, Kirill A. Smirnov1, 2, Boris V. Afanasiev1,3, Olga V. Galkina1, 2, Alexey V. Smirnov1, 2 Prof. Vladimir A. Dobronravov, MD, PhD, DSc, Research Institute of Nephrology, The First St. Petersburg I. Pavlov State Medical University, Russia, L.Tolstoy St. 17, 197089, St. Petersburg, Russia

Subclinical tubular epithelium damage and acute kidney injury following allogeneic hematopoietic stem cell transplantation