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Tel: +98-21-84902473 Fax: +98-21-84902473 Email: p.reyhane@Gmail.com Introduction: Tooth enamel is a precious and highly mineralized tissue in the human body. Amelogenesis Imperfecta (AI) is a developmental, evolutionary and hereditary disease presents with the rare abnormal formation of enamel that affects the primary and secondary dentition. The molecular base of the incomplete quinizium together with clinical manifestation suggest that AI may result from mutations in the FAM83H and ENAM genes. In this study, we aimed to evaluate the association between Amelogenesis Imperfecta and mutations in the FAM83H and ENAM genes in 18 Iranian families with AI in dominant and non-syndromic form.

Determination of mutation in the coding regions of FAM83H and ENAM genes in patients with imperfect enamel (Amelogenesis Imperfecta)