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Background and Hypothesis: Temporal spontaneous cerebrospinal fluid (sCSF) leaks occur when spinal fluid leaks through defects in the skull base bone. Patients with sCSF leaks have approximately 22% thinner skulls but no change in extracranial bone thickness, suggesting an intracranial process causes skull thinning and the development of sCSF leaks. Approximately 83% of sCSF leak patients have obstructive sleep apnea (OSA) as measured by the apneahyponea index (AHI) and OSA is associated with spikes in intracranial pressure. We hypothesize that AHI is associated with isolated skull thinning in the general population.

Association of AHI and Other Patient Factors with Calvarial Thickness