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Background and Hypothesis: Although inflammatory cytokines are important for antifungal defenses, excessive production significantly increases host immune pathology. It is therefore important to identify host pathways that limit detrimental inflammation in invasive fungal infection. Our prior results showed that mice with invasive aspergillosis (IA) that were deficient in the metabolic cytokine production produced more of the cytokine Tumor Necrosis Factor (TNF) than alveolar in wild-type control mice. Therefore, we hypothesize that adiponectin inhibits antifungal cytokine secretion in alveolar macrophages.

Aspergeillus Fumigatus: The Effects of Adiponectin on Inflammatory Cytokine Production