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Background and Hypothesis: Chronic exposure to cigarette smoke (CS) induces DNA lesions and oxidative damage leading to the development of lung diseases such as emphysema and lung cancer. Xeroderma Pigmentosum group C (XPC) is a protein which repairs CS bulky DNA lesions and oxidative DNA damage, including 8-OHdG. We hypothesize that in vitro CS exposure will result in increased 8-OhdG and that XPC knockdown (KD) leads to higher levels of 8-OHdG.

proceedings of imprs

Quantifying Cigarette Smoke Induced Oxidative DNA Damage of Wild Type &amp; XPC Knockdown Human Bronchial Cells Using Human 8-oxoguanine DNA Glycosylase 1