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Primary hyperparathyroidism (PHPT) is unregulated hyperproduction of parathyroid hormone (PTH), inadequate to serum calcium levels. High PTH levels are associated with hypercalciemia, hypercalciuria and hyperphosphaturia, increased bone absorbtion with decreased bone mass and density, gastric hyperacidity with the development of peptic ulcers, and tissue deposition of calium phosphate with pancreatitis, nephrocalcinosis and vascular calcifications. The kidney involvement in PHPT is associated with: nephrolithiasis, nephrocalcinosis, chronic renal disease, and renal tubular dysfunction. DOI: 10.18044/Medinform.201742.653 Original Article MedInform I S S U E 2 , 2 0 1 7 654 MedInform We present a series of 12 consecutive patients (4 male and 8 female, mean age 52.8 +/15.1 years, range 30-73) with kidney damage due to PHPT, admitted to the Clinic of Nephrology for the period May 2013 – September 2017. All patients had nephrolithiasis or history of nephrolithiasis, and nephrocalcinosis (cortical and/or medullary), with or without other organ involvement related to persistent hypercalciemia. We discuss the current knowledge on the diagnosis and treatment of renal involvement in PHPT.

Clinical, laboratory and ultrasound characteristics of patients with primary hyperparathyroidism with renal involvement