Recognizing the fundamental flaw in our dietary studies investigating the impact diets, drugs and lifestyle have on preventing or reversing CAD

Since the introduction of the (a) Inflammation and Heart Disease, and (b) Angina, Theories in the mid-1990s, a transition from a cholesterol only etiology for coronary artery disease (CAD) has taken place; resulting in an understanding that CAD is the result of an Inflammatory process precipitated by a number of factors-including, but not limited to LDL cholesterol and saturated fat [1-5], which impair coronary blood flow.