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Henoch-Schönlein purpura (HSP) is a multisystem, small vessel, leucocytoclastic vasculitis. It is predominantly a childhood vasculitis, rarely reported in adults. Studies have shown that several different genetic factors such as genes involved in inflammatory system and renin-angiotensin system (RAS) are important in the pathogenesis of Henoch-Schönlein purpura.

advances in clinical and experimental medicine

Association of ACE, VEGF and CCL2 gene polymorphisms with Henoch–Schonlein purpura and evaluating the possible interaction effects of these loci in HSP patients