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(ANCA)-associated vasculitis. This report is the was 83% on room air. The abnormal results of the ancillary tests were elevated erythrocyte sedimentation rate and positivity for cytoplasmic-ANCA, together with pulmonary embolism (PE), pulmonary consolidation consistent with PE-related pulmonary infarction, and deep venous thrombosis (DVT), as shown in Figure 1b. Consequently, the patient received a diagnosis of active ANCA-associated vasculitis with VTE (without DAH). She was started on enoxaparin (1 mg/kg twice a day), prednisone (60 mg/day), oral cyclophosphamide, sulfamethoxazole, and oxygen (2 L/min, nasal catheter). There was subsequent clinical improvement. However, two days later, the patient died suddenly, and the circumstances of her death were consistent with the occurrence of another PE. The autopsy revealed DVT/PE without DAH or myocardial infarction.The most important message of this report is that pulmonologists should be alert to the possibility of VTE in ANCA-associated vasculitis. Early clinical suspicion of VTE will avoid delays in diagnosis and treatment, thereby promoting better clinical results. Classically, when hemoptysis does occur (in ANCA-associated vasculitis), “it is usually related to nodules or localized infiltrates, rather than DAH”, which does not include PE as a cause of hemoptysis in ANCA-associated vasculitis.

Incidência de tromboembolismo venoso fatal em vasculite associada a anticorpo anticitoplasma de neutrófilos