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venom is a potent sodium channel activator resulting in autonomic storm (4). Iberitoxin content of venom inhibit the calcium-dependent potassium channels (17). Clinical manifestations, such as vomiting, profuse sweating, priapism in male, cool extremities, hypertension, hypotension, bradycardia, tachycardia, ventricular premature ventricular contraction, transient runs oftachycardia, transient coronary sinus rhythm, pulmonary edema accompanied by marked tented T-waves, ST segment depression, acute myocardial infarction-like pattern, left bundle branch block, and prolongation of QTc interval (400-650 milliseconds). Clinical effects caused by venoms are due to the release of autopharmacological agents into the circulation (4,13). Pharmacokinetic analysis of experimental data on the venom distribution showed that its uptake by the tissue compartment is rather rapid, with estimated half-life of 5.6 minutes; its concentration showed a continuous rise, reaching a peak level within 37 minutes (14). It is interesting to note that, irrespective of different species of scorpions, similar clinical manifestations have been reported (2,11). It confirms that the lethal fraction of venom from different species of scorpion has similar effects on the autonomic nervous system.

Management of severe scorpion sting at rural settings: what is the role of scorpion antivenom?